Its primary mechanism is believed to pivot around enhancing lipolysis, the breakdown of fat, and suppressing lipogenesis, the process of fat accumulation [More et al., 2014]
It works intracellularly by inhibiting NNMT to preserve NAD+ and shift adipocyte metabolism a completely different mechanism, with different research read-outs
AHK-Cu has been studied extensively in animal and laboratory models by the cosmeceutical industry for its effects on skin health, collagen synthesis, and hair growth
Selective NNMT inhibitors (including 5-Amino-1MQ) lower 1-MNA, increase NAD and SAM, suppress adipocyte lipogenesis, and reverse HFD-induced obesity in mice [1]
After reconstitution: store the solution at 28 C (refrigerated) and protect it from light
Oral is the standard